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動物間歇低氧實驗系統

簡要描述:塔望科技提供全系列的動物實驗用低/高氧控制產品,包括恒定濃度控制的低氧動物箱、高氧動物箱、可編程的間歇氧濃度控制系統、帶緩沖艙的手套低氧箱等。整套間歇氧氧實驗箱裝置主要由氧氣控制器和動物實驗箱兩部分組成。另可提供多種不同的氣體控制器,滿足不同實驗O2、CO2、NO、CO、O3等氣體濃度控制的需求。

  • 產品型號:ProOx-100
  • 廠商性質:生產廠家
  • 更新時間:2026-01-20
  • 訪  問  量:2924

詳細介紹

品牌其他品牌產地類別國產
應用領域醫療衛生,生物產業,制藥/生物制藥

產品描述

塔望科技提供全系列的動物實驗用低/高氧控制產品,包括恒定濃度控制的低氧動物箱、高氧動物箱、可編程的間歇氧濃度控制系統、帶緩沖艙的手套低氧箱等。整套間歇氧氧實驗箱裝置主要由氧氣控制器和動物實驗箱兩部分組成。另可提供多種不同的氣體控制器,滿足不同實驗O2、CO2、NO、CO、O3等氣體濃度控制的需求。

ProOx-100動物間歇低氧實驗系統可以控制動物實驗箱內持續低氧的環境,用以制造相關的間歇低氧實驗模型。用戶可自由設置所需要的濃度和實驗持續時間,所有的設置通過控制主機觸摸屏完成,人性化設計,操作簡便。

ProOx-100動物間歇低氧實驗系統監測指標全面,動物低氧艙內具有集成化的傳感器模塊,內置溫度、濕度、氧氣、二氧化碳傳感器。可以實時監測動物低氧艙內的環境。系統通過閉環反饋控制,根據動物低氧艙內的氧濃度實時反饋控制,使動物實驗數據更準確。Ox-100動物低氧實驗系統具有優良的控制性能,持續低氧實驗時,氧濃度的誤差為0.1%

如需高氧實驗,請選擇型號Ox-100HE


產品特點及參數

1. 為動物間歇低氧實驗模型的建立提供穩定的低氧環境

2. 按照設定氣體濃度自動配比氣體,無需在箱體外混合比例氣體,實驗氧濃度的準確,節省氣源

3. 艙體采用全透明PMMA材質,防止由于光線影響動物生物節律

4. 7英寸大屏觸摸屏控制,人性化界面,操作簡單

5. 監測參數:溫度、濕度、氧氣O2濃度、二氧化碳濃度

6. 控制精度:±0.1%

7. 非色散紅外(NDIR)二氧化碳傳感器,測量范圍:05000ppm

8. 進口電化學氧氣O2濃度檢測器,測量范圍:0-25%vol,線性度好,檢測準確、使用壽命長。具有溫度補償機制

9. 溫度檢測:進口高精度溫度傳感器

10. 氧氣濃度變化動態曲線,直觀了解氧氣濃度變化的過程

11. 具有定時功能,實驗完成,自動恢復常氧狀態,并伴有聲音提示

12. 氧氣濃度自動校準:通過控制器對傳感器快速校準

13. *的氣體混合及循環機制,保證箱體內氣體濃度的均一

14. 高性能電磁閥,性能穩定,超長壽命

15. 艙體尺寸有多種選擇,可靈活搭配。也可根據實驗要求進行定制



ProOx-100動物間歇低氧實驗系統多功能控制


可進行間歇低氧實驗(CIH)、急性缺氧實驗、慢性缺氧實驗、高氧/低氧交替實驗



動物間歇低氧實驗系統












應用領域

肺動脈高壓、腎臟疾病研究、腫瘤研究、心血管疾病研究、視網膜病變、運動醫學研究、OSAHS、腦發育與神經生物學、干細胞研究、醫學研究等


型號說明


名稱

型號

說明

單位

動物低氧實驗系統

Ox-100

恒定氧控制,低氧

動物氧濃度實驗系統

Ox-100HE

恒定氧控制,低氧/高氧

動物間歇低氧實驗系統

ProOx-100

恒定氧控制/間歇氧控制,低氧

動物間歇氧濃度實驗系統

ProOx-100HE

恒定氧控制/間歇氧控制,低氧/高氧

動物間歇低氧實驗系統ProOx-100-CIH

專用于睡眠呼吸暫停造模


艙體型號(可選擇不同尺寸的低氧艙)

名稱

型號

說明

單位

動物實驗艙體小號

OxC-S

大鼠籠x1

動物實驗艙體中號

OxC-M

大鼠籠x2

動物實驗艙體大號

OxC-L

大鼠籠x4

動物實驗艙體特大號

OxC-XL

大鼠籠x8

動物實驗艙體-CIH

OxC-CIH

36只小鼠

手套操作箱

Gl-700

700L



引用文獻


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[40] Feng X, Li C, Zhang W, et al. Mechanism of retinal angiogenesis induced by HIF-1α and HIF-2α under hyperoxic conditions[J]. Scientific Reports, 2025, 15(1): 36049.

[41] Yao Y, Chen Y, Li Y, et al. TGM2 Enhances Hypobaric Hypoxia-mediated Brain Injury Via Regulating NLRP3/GSDMD Signaling[J]. Neurochemical Research, 2025, 50(6): 1-11.

[42] Yang A, Guo L, Zhang Y, et al. MFN2-mediated mitochondrial fusion facilitates acute hypobaric hypoxia-induced cardiac dysfunction by increasing glucose catabolism and ROS production[J]. Biochimica et Biophysica Acta (BBA)-General Subjects, 2023: 130413.

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